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Fat cell deficiency can lead to muscle and liver insulin resistance

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According to U.S. researchers reported that not a normal intake of fat cells, glucose triggers the muscles and liver occurred the same problem.

Boston, Beth Israel Deaconess Medical Center researchers found that fat cells in mice to eliminate the transfer of manipulation of insulin in the blood glucose to enter cells, a key protein, the loss of muscle glucose uptake of insulin signaling in promoting the reaction, while the liver also showed an organ of glucose production of insulin to

Insulin-resistant lipid-lowering efficacy

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Second People's Hospital of Nanning, Guangxi physicians in clinical practice found that the merger of insulin resistance in patients with hyperlipidemia, simple use of lipid-lowering medication, and often does not receive its due effect.

Traditional treatment for hyperlipidemia is dietary adjustment, weight control, appropriate exercise, oral statins or fibrates lipid-lowering medication. Nanning second hospital five general practitioners found that some

Diabetes should be an early use of insulin

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Many patients with type Ⅱ diabetes mellitus are unwilling to accept insulin therapy, fear of addiction, or cause adverse reactions. This view is wrong. Β-cell function in patients with diabetes is a problem only need to add. In Western developed countries, more than 50% of type Ⅱ diabetes mellitus patients received insulin therapy, while in China, this proportion is only about 10% of diabetic patients are not in fact the Chinese do not need to use insulin, but the understanding of

“Long-lasting” alternative can not be “short-acting”

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In many diabetic patients the impression that, if the bad effect of using short-acting insulin, it is necessary to use the efficiency or long-acting insulin, in fact, this argument is wrong. Because different formulations of insulin, treatment onset time, duration of efficacy is different in usage and can not be replaced.

The slow onset long-acting insulin, but the relatively long duration of efficacy, simple to say, is the ability to control blood sugar a long time, while the

Insulin-sensitizing agent merits and demerits of

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What is insulin resistance?

Insulin resistance refers to the role of insulin organs (target organs) - mainly in skeletal muscle, adipose tissue and liver to insulin sensitivity and reactivity of reduced or lost, and therefore lower than the normal amount of insulin produced physiological effects, or the need to achieve normal physiological effects of higher than normal amount of insulin, a phenomenon known as insulin resistance. Insulin resistance occurs